</p> 第 4部分:阅读理解 (第 31~45题,每题 3分 ,共 45分)! z# N0 ^1 E. F- T6 O5 |: m
下面有3篇短文,每篇短文有5道题。请根据短文内容,为每题定1个最佳选项。
9 n+ s$ u+ z/ u3 L: T9 V1 s 第一篇
8 _9 g' x# s$ _ Human Heart Can Make New Cells0 M7 _9 ?0 ~. ^7 h+ i
Solving a longstanding mystery, scientists have found that the human heart continues to generate new cardiac cells throughout the life span, although the rate of new cell production slows with age.
! W' S4 b2 ~5 {& } The finding, published in the April 3 issue of Science, could open a new path for the treatment of heart diseases such as heart failure and heart attack ,experts say.
" S' r0 i& S) H! v R "We find that the beating cells in the heart, cardiomyocytes, are renewed, " said lead researcher Dr. Jonas Frisen, a professor of stem cell research at the Karolinska Institute in Stockholm, Sweden. "It has previously not been known whether we were limited to the cardiomyocytes we are bom with or if they could be renewed." he said.0 y: i& H9 U2 t i' f# g( i( V4 o
The process of renewing these cells changes over time, Frisen added. In a 20-year-old, about 1 percent of cardiomyocytes are exchanged each year, but the turnover rate decreases with age to only 0. 45 percent by age 75.
7 z# l. w8 K! ?, Q7 J8 Y "If we can understand how the generation of new cardiomyocytes is regulated , it may be potentially possible to develop pharmaceuticals that promote this process to stimulate regeneration after for example, a heart attack, " Frisen said.
) u, i$ r& `3 O, w% [ That could lead to treatment that helps restore damaged hearts.
& v- u' z4 }/ e, D$ [; G "A lot of people suffer from chronic heart failure," noted co-author Dr. Ratan Bhardwaj.
5 K/ k T- C+ `2 |' y9 u! e2 i Ratan Bhardwaj, also from the Karolinska Institute. " Chronic heart failure arises from heart cells dying, "he said.
3 o$ ]& G8 B4 | With this finding, scientists are "opening the door to potential therapies to having ourselves heal ourselves, " Bhardwaj said. " Maybe one could devise a pharmaceutical agent that would make heart cells make new and more cells to overcome the problem they are facing."7 s5 s' [9 T& \6 n! n
But barriers remain. According to Bhardwaj , scientists do not yet know how to increase heart cell production to a rate that would replace cells faster than they are dying off, especially in older patients with heart failure. In addition, the number of new cells the heart produces was estimated using healthy hearts-whether the rate of cell tumover in diseased hearts is the same remains unknown.
8 r: Z4 R& z/ Q4 k* P/ W. d 31. The human heart stops producing cardiac cells1 u) U' l1 z" L' R# X D6 c6 [
A. when a person becomes old
" G/ k7 ^6 K5 u, h$ r B. as soon as a person gets sick' {+ T, ^: y. s D. b1 r
C. immediately after a person is born
, |1 p$ W! q: v# T7 {; E& I D. once a person dies# s/ h. r! j& f& \
32. The finding could prove to be useful to
, x/ H y1 i: ]: Y A. the analysis of cardiac cells* s Z* }4 ]% R& k+ T3 Y
B. the prevention of chronic diseases
+ l6 a+ o- }& G C. the treatment of heart disease! i9 D* C# K. a( ^
D. the study of longstanding mysteries/ d9 M: z2 q% f9 k+ w
33. In people in their mid-70s, only 0. 45 percent of cardiomyocytes _ .
; c% G6 Y( A0 L4 } @" x A. are still functional" f& V9 e6 n' K X
B. are reduced each year7 a! o! _6 o2 b$ u
C. are replaced each year
) s) k$ J# V3 S% q4 \% o. s D. are damaged each year e& e: t* B$ ]8 m" U4 V+ w
34. Chronic heart failure is attributed to4 B1 u& I8 E' v
A. the dying heart cells, k7 R: i+ T- u! {) X
B. the effect of pharmceutical4 s2 K6 v6 b; L0 U. ^( c
C. the weight of a patient
& l9 Z& D- k. N% l2 _1 S D. the life span of a person
# ]0 f9 o6 u$ z7 }0 X1 F- ?' H0 H; ? 35. It is not known yet if the rate of cell turnover in diseased hearts
/ i1 J+ {, j a A. is high enough to replace cells faster than they're dying off. y$ b1 D v% n# v' q
B. is of any use to researchers3 ^$ }0 c8 O. S4 ]
C. is the same as that in healthy hearts! Z( W' \$ p# M6 ], ^1 g# P
D. changes over time |